Bladder pain in BPS/IC may result from an exaggerated response of TRPV1 and enhanced urothelial ATP release induced by chronic adrenergic overactivity

Research Type

Pure and Applied Science / Translational

Abstract Category

Pelvic Pain Syndromes

Links
	Abstract 444
	Nerves and Bladder Sensations Scientific Podium Session 29
	Thursday 8th October 2015
	15:30 - 15:45
	510C
	Painful Bladder Syndrome/Interstitial Cystitis (IC) Pharmacology Basic Science
	1. IBMC – Instituto de Biologia Molecular e Celular, University of Porto, Porto, Portugal, 2. Group of animal genetics evolution, CIIMAR, University of Porto, Porto, Portugal, 3. Department of Continence Medicine, University of Tokyo, Tokyo, Japan, 4. Departament of Urology, Hospital São João, Porto, Portugal; Department of Renal, Urologic and Infectious diseases, Faculty of Medicine of University of Porto, Porto, Portugal; IBMC – Instituto de Biologia Molecular e Celular, University of Porto, Porto, Portugal, 5. IBMC – Instituto de Biologia Molecular e Celular, University of Porto, Porto, Portugal ; Department of Renal, Urologic and Infectious diseases, Faculty of Medicine of University of Porto, Porto, Portugal
Presenter
A Ana Charrua IBMC – Instituto de Biologia Molecular e Celular, University of Porto, Porto, Portugal ; Department of Renal, Urologic and Infectious diseases, Faculty of Medicine of University of Porto, Porto, Portugal
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